Dr. Shehab Al-Abed - Medical Retina & Uveitis

Dr. Shehab Al-Abed - Medical Retina & Uveitis Medical Retina & Uveitis Consultant 🇯🇴 | Software Developer 💻 | Founder of Retina Squad Academy 👁️ | Simplifying ophthalmology for the globe.

01/08/2026

The eye is no longer just about vision. It is the ultimate biomarker. 👁️💻

​🔥 THE ONE FACT PEARL:

"Oculomics" is the rapidly growing field that uses Artificial Intelligence to extract systemic health data from ophthalmic imaging. The retinal microvasculature provides a direct, non-invasive look at the body's vascular and neurological health. With AI, a single fundus photograph can now predict cardiovascular events, assess diabetic progression, and even screen for neurodegenerative diseases like Alzheimer's.

​I am incredibly excited to announce that I will be speaking about this exact topic—bridging the gap between clinical ophthalmology and AI data pipelines—at the 10th International Conference of the Faculty of Medicine: "Advancing Clinical Excellence and Innovation in Healthcare" on August 19-20.

​We will be discussing the Jordan A-Eye model, Uveitis DDx classifiers, and how we are building the future of digital health.

​Drop a 🤖 in the comments if you are fascinated by medical AI, and let me know if I will be seeing you at the conference!

31/07/2026

Swollen disc? Don't panic. 🛑👁️

​Optic Disc Drusen (ODD) can perfectly mimic papilledema, leading to unnecessary anxiety and expensive neurological workups.

​🔥 THE ONE FACT PEARL:

Because drusen contain calcium, they exhibit natural hyperautofluorescence. A quick, non-invasive FAF scan in the clinic will immediately light them up, confirming the diagnosis of pseudopapilledema.



Case images from DOI:10.3389/fmed.2021.735805

17/07/2026

Why do we call them "Bone Spicules"? 🦴👁️⚡

​We see this classic sign in Retinitis Pigmentosa all the time, but the origin of the name is a beautiful example of how pathology mimics anatomy.

​As photoreceptors undergo apoptosis in RP, the RPE cells migrate into the inner retina and deposit melanin in a perivascular pattern. This branching network looks exactly like the trabeculae (spicules) found in cancellous bone under a microscope.

​🔥 HIGH-YIELD FEBOS-R PEARL:

Remember that this pigment is not just sitting randomly in the retina. The RPE cells actively migrate and accumulate around the retinal vessels. Over time, this contributes to the classic "vascular attenuation" or narrowing of the vessels we see as part of the classic RP triad (Bone spicules, waxy disc pallor, vascular attenuation).

​Drop a 🦴 in the comments if you love connecting the clinical dots!

انا من داعمي منصة علم لاني اؤمن بان العلم يجب ان يكون للجميع حتى يدعم التطور والبحث الاكاديمي والسريري
15/07/2026

انا من داعمي منصة علم لاني اؤمن بان العلم يجب ان يكون للجميع حتى يدعم التطور والبحث الاكاديمي والسريري

منصة "علم"
ليست مجرد مكتبة طبية، بل مرجع علمي موثوق يواكب أحدث المستجدات الطبية، ويساعد على تطوير الممارسة السريرية واتخاذ قرارات مبنية على الأدلة. 📚🩺
📥حمّل التطبيق الآن:

App store: ELM – Jordan
▶ IOS: https://apple.co/3uQdsyV
📱Google play: ELM – Jordan
▶ Android: https://bit.ly/3uNECXa




10/07/2026

Not all CME leaks. Do you know the differential for angiographically silent edema? 👁️⚡

​When a patient presents with massive cystic spaces in the INL and ONL on OCT, our first instinct is to look for the classic petaloid leakage on FFA. But what happens when the macula is dark, yet the RPE shows widespread window defects?

​Your differential has to shift entirely.

​🔥 HIGH-YIELD FEBOS-R PEARL:
The causes of angiographically "silent" or non-leaky CME include:

1️⃣ Nicotinic Acid (Niacin) Toxicity
2️⃣ Taxane Toxicity (Paclitaxel/Docetaxel)
3️⃣ X-linked Retinoschisis (XLRS)
4️⃣ Retinitis Pigmentosa (RP)
5️⃣ Goldmann-Favre Syndrome

​In this case (an EYS-related autosomal recessive Retinitis Pigmentosa), the CME is driven by RPE pumping failure and Müller cell dysfunction rather than primary vascular breakdown. The Fundus Autofluorescence (FAF) reveals the true extent of the disease with a classic hyperautofluorescent macular ring and peripheral RPE atrophy.

​References:

1. ​Strong, S., et al. (2017). Cystoid macular oedema in retinitis pigmentosa: pathogenesis and rationale for therapy. British Journal of Ophthalmology.

2. ​Liew, G., et al. (2019). Pathogenesis of cystoid macular edema. Survey of Ophthalmology.

​ CME

27/06/2026

Type 1 MNV on OCT-A.. This is part of the full length lecture.. Check pinned comment for link to post our visir my page!

24/06/2026

Masterclass OCT-A lecture is out.. Detailed and up-to-date information..

Check pinned comment below
👇👇👇

19/06/2026

🚨 No needles. No dye. Just pure vascular mapping. Welcome to the OCT-A Revolution. 👁️⚡

​In Lecture 1.2 of the Retina Squad Academy, we are breaking down the complete framework you need to master this technology in the clinic and confidently crush the FEBOS-R exam.

​Here is what we are decoding today:
- ​The 4 Vascular Plexuses: Pinpointing the SCP, ICP, DCP, & Choriocapillaris.
- ​MNV Types 1, 2, and 3: How to recognize their specific flow patterns instantly.
- ​The FEBOS-R and boards Traps: How to spot projection artifacts that fake disease.
- ​OCT-A vs. FFA: The definitive guide on what each modality can (and cannot) tell you.

​👇 Question for the Retina community: Do you find yourself relying more on OCT-A or traditional FFA when diagnosing complex MNV? Let’s debate in the comments!

​☕ A special thank you to my new Page Supporters! you too can join the inner circle and grab your custom badge.

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Amman

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